Subclinical pertussis infection, which results from the vaccine’s failure to produce mucosal immunity and prevent infection and transmission, is believed to be a direct cause of MS via impaired immunity to pertussis, which is a neurotoxin.

PMID: 

Immunobiology. 2016 Apr ;221(4):512-5. Epub 2015 Dec 18. PMID: 26724970

Abstract Title: 

The potential role of subclinical Bordetella Pertussis colonization in the etiology of multiple sclerosis.

Abstract: 

It is established that (1) subclinical Bordetella pertussis colonization of the nasopharynx persists in highly vaccinated populations, and (2) B. pertussis toxin is a potent adjuvant that, when co-administered with neural antigens, induces neuropathology in experimental autoimmune encephalomyelitis, the principle animal model of multiple sclerosis. Building on these observations with supporting epidemiologic and biologic evidence, we propose that, contrary to conventional wisdom that subclinical pertussis infections are innocuous to hosts, B. pertussis colonization is an important cause of multiple sclerosis.

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